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Table 1 Demographic and neuropsychological features in single EOAD patient

From: The combined effects of microglia activation and brain glucose hypometabolism in early-onset Alzheimer’s disease

 EOAD1EOAD2EOAD3EOAD4EOAD5EOAD6EOAD7EOAD8EOAD9EOAD10EOAD11EOAD12
Age, years685556645859645854566366
Education, years5131381388881385
SexFMMFFMFFMFFM
Disease duration, years511321151232
DiagnosistADtADtADtADfADfADfADfADfADfADPCAPCA
MMSE202117221810121817222121
Token test313123.5292311.519.2522.25333127.530.5
PVF271072916631322315644
SVF30.1818.542.8121681345.54429.259.2
Span forward6.394.842.835.135.756.132.925.042.925.834.136.02
Span backward4.53< 3< 33.19< 3< 3< 33.1< 33.794.19< 3
RAVLT IR22.114.813.824.310UTUT0364121.532.3
RAVLT DR00001.5UTUT04.2433.6
ROF recall8UT08.55.25UT055.7511.7507.5
ROF copy9UT02313.2537.2528.529.2514.75024.25
CDT110255000107010
Attentive Matrix36UT12.531.2523UT17.523224934.2523
Stroop time33.52844.2536.7529.25UTUTUT46.846.1744.2527
Stroop errors9.53.51311.2526.5UTUTUT505.252.5
Raven PM19.5UT1419UTUTUT21.526.5221231.5
  1. EOAD early-onset Alzheimer disease, AD Alzheimer disease, tAD typical AD, fAD frontal Alzheimer disease, PCA posterior cortical atrophy, MMSE mini mental state examination, PVF phonological verbal fluency, SVP semantic verbal fluency, RAVLT Rey auditory verbal learning task, IR immediate recall, DR delayed recall, ROF Rey-Osterrieth figure, CDT clock drawing test, Raven PM Raven Progressive Matrices, UT untestable
  2. For neuropsychological assessment, pathological scores are reported in bold