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Table 1 Human mediotemporal gyrus samples used in this study

From: Sorting nexin-4 regulates β-amyloid production by modulating β-site-activating cleavage enzyme-1

Diagnosis

Sex

Age (years)

Braak stage

Amyloid

PMD (h:minutes)

pH

Weight (g)

Alzheimer’s disease

M

85

5

C

07:10

6.13

1020

Alzheimer’s disease

M

65

6

C

08:50

6.88

1057

Alzheimer’s disease

M

65

5

C

05:50

6.36

1355

Alzheimer’s disease

M

65

5

C

07:20

6.47

1173

Alzheimer’s disease

M

87

5

C

06:10

6.14

1047

Alzheimer’s disease

M

67

5

C

04:10

6.40

1252

Alzheimer’s disease

M

70

6

C

04:50

6.95

1040

Alzheimer’s disease

M

82

5

C

05:15

6.34

1182

Nondemented control

M

73

0

O

24:45

?

1267

Nondemented control

M

71

1

O

07:40

6.20

1150

Nondemented control

M

87

1

A

10:20

6.32

1256

Nondemented control

M

80

0

O

07:15

5.80

1331

Nondemented control

M

84

1

A

05:35

6.98

1337

Nondemented control

M

82

1

O

05:10

6.75

1087

Nondemented control

M

78

1

O

<17:40

6.52

1125

  1. Mediotemporal gyri from eight patients with AD and seven age- and sex-matched controls were provided by the Netherlands Brain Bank. Braak stages based on neurofibrillary tangles were 5 or 6 in subjects with Alzheimer’s disease (AD) and 0 or 1 in the controls [24]. Braak stages based on amyloid plaques were C in AD cases and 0 or A in the controls [24]. Tissue preparation time from death is displayed as postmortem delay (PMD)
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Alzheimer's Research & Therapy

ISSN: 1758-9193