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Table 1 Human mediotemporal gyrus samples used in this study

From: Sorting nexin-4 regulates β-amyloid production by modulating β-site-activating cleavage enzyme-1

Diagnosis Sex Age (years) Braak stage Amyloid PMD (h:minutes) pH Weight (g)
Alzheimer’s disease M 85 5 C 07:10 6.13 1020
Alzheimer’s disease M 65 6 C 08:50 6.88 1057
Alzheimer’s disease M 65 5 C 05:50 6.36 1355
Alzheimer’s disease M 65 5 C 07:20 6.47 1173
Alzheimer’s disease M 87 5 C 06:10 6.14 1047
Alzheimer’s disease M 67 5 C 04:10 6.40 1252
Alzheimer’s disease M 70 6 C 04:50 6.95 1040
Alzheimer’s disease M 82 5 C 05:15 6.34 1182
Nondemented control M 73 0 O 24:45 ? 1267
Nondemented control M 71 1 O 07:40 6.20 1150
Nondemented control M 87 1 A 10:20 6.32 1256
Nondemented control M 80 0 O 07:15 5.80 1331
Nondemented control M 84 1 A 05:35 6.98 1337
Nondemented control M 82 1 O 05:10 6.75 1087
Nondemented control M 78 1 O <17:40 6.52 1125
  1. Mediotemporal gyri from eight patients with AD and seven age- and sex-matched controls were provided by the Netherlands Brain Bank. Braak stages based on neurofibrillary tangles were 5 or 6 in subjects with Alzheimer’s disease (AD) and 0 or 1 in the controls [24]. Braak stages based on amyloid plaques were C in AD cases and 0 or A in the controls [24]. Tissue preparation time from death is displayed as postmortem delay (PMD)